Upper Cervicogenic Dysfunction That (upper) cervicogenic dysfunction does not play a causal role in primary headache (i.e. migraine, tension and cluster headache, hemicrania continua, menstrual migraine etc) syndromes – wrong! Research continues to support the concept that the many and varied forms of headache and migraine1-7 – migraine, tension-type headache, cluster headache (that takes care
And Cervicogenic Headache A recent (and frequent) query from a colleague(s): “I wonder if general practitioners are aware of that (i.e. the natural progression of cervicogenic (neck) headache… If it is left untreated, cervicogenic headache becomes more frequent, more severe, requiring stronger and stronger medication, and eventually becomes continuous see Cervicogenic Management Decreases Migraine Progression). I
CGRP Under the Magnifying Glass There is now widespread agreement that vasodilation is neither necessary nor of a magnitude to be responsible for migraine pain; migraine is a complex neuronal disorder with vascular epiphenomenon.1 Interest in calcitonin gene-related peptide (CGRP) comes from research which suggests that CGRP could play an important role of migraine pathophysiology.
Cervicogenic Headache Headache is classified as either Primary or Secondary Headache.1 Primary Headaches are those headache and migraine conditions with unknown pathophysiology. Secondary Headache comprises headache arising from a recognised and accepted cause, i.e. secondary to a known source. Cervicogenic Headache (CeH) is classified as a secondary headache i.e. headache secondary to a cervical lesion.1
When Managing Headache and Migraine A colleague recently sought to question some of my criticism of medical diagnosis in migraine, see Commentary for Health Professionals, ‘Managing Headache and Migraine: Why Treatment Often Fails‘. My thoughts on my colleague’s comments/queries (in bold italics) follow. Yes there is no test for migraine so the diagnosis is based
In Trigeminal Autonomic Cephalalgias It is refreshing to come across a case study which supports my clinical experience. This study1 describes a woman with a cluster-like pattern of presumably [R] side-locked face and neck pain with associated periorbital and mandibular swelling, tearing, conjunctival injection, and allodynia which was ameliorated by third occipital nerve lesioning –
